Scientists were able to stimulate mice to eat less or more than usual by influencing a mechanism in their
During their work, the authors noticedthat the activity of these groups of DA-VTA neurons, which produce dopamine, and DRD1-LPBN, which regulate food intake, increases sharply before the mice finish eating. Then the researchers tried to interfere with the operation of this mechanism.
Biologists suppressed neuron activity as a resultThe mice began to eat to their fill. Conversely, artificial stimulation of these neurons discouraged mice from any desire to eat. This means that the effect on the brain prevented the satiety response.
According to scientists, based on the data of the researchers, it will be possible to create drugs that help people in the fight against obesity.
The authors suggest that in humans, the satiety sensory system works in a similar way, since the low-level neural mechanisms are often the same.
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