In a new study, Professor Jane Lucas of the University of Southampton (UK) and her
SARS-CoV-2 invades the lung cells of humans and other mammals using ACE2 receptor molecules that coattheir surface, so it is relevant to study all the mechanisms of its effect on the body.
Biologists have found in the nasal mucosaan unusual variety of this protein. It was two times shorter than its classic form, and inside it did not have a segment to which the coronavirus attaches itself.
At first, we were just very happy to have found a new form of ACE2.However, learning that it can prevent the spread of the coronavirus is not only where it penetratesinto the human body, but also in other areas of the respiratory tract, we became even more interested in the find.
Jane Lucas, Professor, University of Southampton in the UK
Further experiments showed that the cells of the nasal mucosa begin to produce similar molecules in responseCells begin to produce them after the onset of a cold, but they react to COVID-19otherwise.
This means that the rate at which SARS-CoV-2 spreads through the respiratory tract can be drastically reduced if the mucosal cells are forced to produce a truncated ACE2 protein rather than its full form.
Now, the scientists are going to conduct experiments on different animals to see if it works in a similar wayScientists hope to use it to create a new cure for the disease. COVID-19
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