Scientists figured out how to "reprogram" the heart using an existing drug

Transplantation saves lives, but organs don't last very long. University of Michigan staff

found that an existing drug “reprograms” the donor heart so that it can be stored outside the body longer. This will reduce the risk of organ failure after transplantation.

Currently, donor hearts are stored inrefrigerator for only about four hours. The problem is that a molecule called succinate accumulates in the organ while it is on ice. Later, when blood enters the heart again, this substance causes oxidative stress, which then leads to organ damage and heart failure. It is one of the leading causes of death in transplant patients.

Scientists from the University of Michigan have testedhow to address the problem with itaconate, an enzyme known to neutralize succinate. By reviewing a database of metabolic effects, the team identified valproic acid as a promising source of itaconate.

Using metabolic screening, we found that valproic acid can reprogram the donor heart to produce beneficial itaconate during organ preservation.

Paul Tang, senior author of the study

In tests on human and porcine hearts,when stored in the refrigerator, valproic acid helped the heart produce antioxidants and anti-inflammatory proteins that relieved the stress caused by succinate.

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